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Metabolism and Target Organ Damage (M&TOD) is an international peer-reviewed, open access, online journal.

Aims and Scope

Metabolism and Target Organ Damage (M&TOD) is an international, peer-reviewed, open access interdisciplinary journal which provides an online platform for publication of clinical, basic and translational studies. It covers (cardio)-metabolic disorders per se, such as obesity, diabetes, dyslipidemias, arterial hypertension and hyperuricemia in all age groups. Of note, this journal will emphasize the role of these metabolic disorders as either effectors/amplifiers or consequences of the development and progression of injuries to target organs including but not limited to the cardiovascular system, the liver, kidneys and the pancreas. Specific applications in metabolic disorders are as follows: history of disease, epidemiology, prevention, natural history, genetics, cell and molecular biology, pathobiochemistry, physiopathology, anatomic pathology, clinical chemistry, pharmaceutical chemistry, pharmacology, psychology, psychiatry, statistics and immunology.

From a clinical point of view, the journal covers aspects of biomarkers, diagnosis, prognosis and all types of innovative management strategies including cognitive-behavioral approaches and randomized controlled trials of innovative drug schedules of metabolic disorders. M&TOD welcomes to publish researches to promote the development of animal-free models of studies including cell cultures, artificial intelligence and others.

M&TOD particularly welcomes studies of translational value of ranging from basic science to clinical applications which is interested in evaluating submissions pertaining to all areas of public health including health policy; health economics, internal medicine and its sub-specialties, such as cardiology, endocrinology hepato-gastroenterology, nephrology, oncology, general surgery and its branches such as bariatric, endocrine, vascular, metabolic, oncological surgery.

M&TOD welcomes submissions of the following editorial categories: Original Article, Review, Commentary, Mini-Review, Systematic Review, Meta-analysis, Editorial, Perspective, Opinion, Communication, Letter and Research Highlight.


The journal is owned by OAE Publishing Inc.

Publishing Model

Gold open access. All articles published by M&TOD are made freely and permanently accessible online immediately from the date of publication. For further information, please refer to Open Access.

Copyright and License to Publish

Articles in M&TOD are published under a Creative Commons Attribution 4.0 International (CC BY 4.0). The CC BY 4.0 allows for maximum dissemination and re-use of open access materials and is preferred by many research funding bodies. Under this license users are free to share (copy, distribute and transmit) and remix (adapt) the contribution for any purposes, even commercially, provided that the users appropriately acknowledge the original authors and the source.

Copyright is retained by authors. Authors are required to sign a License to Publish (which can be downloaded from the journal's Author Instructions), granting M&TOD, which identifies itself as the original publisher, exclusive rights to publish their articles, and granting any third party the right to use the articles freely as long as the integrity is maintained and the original authors, citation details and publisher are identified.

Editorial Board

For more information about the editorial team, please refer to Editorial Board.

Editorial Policies

All manuscripts submitted to M&TOD should adhere to M&TOD's Editorial Policies.

Peer Review

The journal adheres to rigorous peer review and undergoes single-blind peer review. For more details, please refer to Editorial Process and Peer Review Guidelines.

Journal Information and Statistics

  • Publication model: Open access
  • Frequency: Quarterly
  • ISSN 2769-6375 (Online)

Indexing & Abstracting

All articles published in M&TOD are included in:


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Metabolism and Target Organ Damage
ISSN 2769-6375 (Online)


All published articles are preserved here permanently:


All published articles are preserved here permanently: